Australia's leading material handling equipment supplier is once again backing a bid to raise research funding for a baffling muscle disease.
Toyota Material Handling Australia (TMHA) is a major sponsor of Save Our Sons, a charitable organisation seeking to raise public awareness of and research funding for Duchenne Muscular Dystrophy (DMD).
The Save Our Sons charity is seeking to fund a human clinical trial with the aim of finding a cure for the disease, which only affects boys.
The highlight of this year's fundraising program is the Save Our Sons Play for a Cure - Casino Royale poker tournament at Sydney's Australian Technology Park on Saturday May 11, hosted by World Poker Champion Joe Hachem.
First prize includes flights to a seat in the World Series of Poker Tournament in Las Vegas and $3000 towards air fares and accommodation.
TMHA executive vice president and COO Steve Takacs said the company was honoured to be part of the Save Our Sons campaign to find a cure for DMD.
"DMD is a terrible disease, with children battling against the clock to stay self-sufficient without the need for a wheelchair and, ultimately, to survive," he said.
Save Our Sons was founded by Sydney train driver Eli Eid and his best mate, contract painter Bass Abboud, after Eid's son was diagnosed with DMD.
Save Our Sons seeks to raise public awareness and much-needed funding for research on the disease.
"We started in February 2008, just the two of us, because I wanted to help a mate who was doing it tough - with his son likely to die before the age of 20," Save Our Sons vice president Bass Abboud said.
DMD is a form of muscular dystrophy characterised by aggressive muscle degeneration, leading to the inability to walk and eventually death due to heart and lung failure.
This disease only affects boys, with symptoms presenting themselves before the age of five.
DMD affects one in every 3500 boys, making it the most prevalent of muscular dystrophies.
Although only males are afflicted, females can be carriers. The disorder is caused by a mutation in the Dystrophin gene.